A novel biochemical marker for predicting the severity of ACS with unstable angina pectoris: Asprosin

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dc.contributor.authorAcara, Ahmet Çağdaş
dc.contributor.authorBolatkale, Mustafa
dc.contributor.authorKızıloğlu, İlker
dc.contributor.authorİbişoğlu, Ersin
dc.contributor.authorCan, Çagdaş
dc.date.accessioned10.07.201910:49:13
dc.date.accessioned2019-07-10T19:50:24Z
dc.date.available10.07.201910:49:13
dc.date.available2019-07-10T19:50:24Z
dc.date.issued2018
dc.departmentİstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, Acil Tıp Ana Bilim Dalı
dc.departmentİstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, Kardiyoloji Ana Bilim Dalı
dc.descriptionWOS: 000439103800039
dc.descriptionPubMed ID: 29274804
dc.description.abstractIschemic Heart Disease (IHD) is the leading cause of death world-wide, accounting for 12.7% of global mortality[1] . Acute Coronary Syn-drome (ACS) covers a wide spectrum of clinical conditions ranging fromunstable angina to Non-ST Elevation Myocardial Infarction (N-STEMI)and ST Elevation Myocardial Infarction (STEMI). New treatments andmanagement guidelines emerge for the care of patients with ACS; riskstratification is the cornerstone in the initial evaluation of these patients[2] . That inflammatory mediators play a role duringthe evolution of ACSis indicated by the widespread coronary inflammation found during un-stable angina pectoris (UAP), throughout theentire coronary artery bed,and in the extent that ACS outcome is related to a concurrent inflamma-toryresponse[3,4] . Circulatingasprosin,a protein hormone, responds tolow dietary glucose by triggering the release of liver glucose stores, andthe reduction of asprosin protects against the hyperinsulinism associat-ed with metabolic syndrome.
dc.identifier.citationAcara, A., Bolatkale, M., Kızıloğlu, İ., İbişoğlu, E. ve Can, Ç. (2018). A novel biochemical marker for predicting the severity of ACS with unstable angina pectoris: Asprosin. American Journal of Emergency Medicine, 36(8), 1504-1505. https://dx.doi.org/10.1016/j.ajem.2017.12.032
dc.identifier.doi10.1016/j.ajem.2017.12.032
dc.identifier.endpage1505
dc.identifier.issn0735-6757
dc.identifier.issn1532-8171
dc.identifier.issue8
dc.identifier.scopusqualityQ1
dc.identifier.startpage1504
dc.identifier.urihttps://dx.doi.org/10.1016/j.ajem.2017.12.032
dc.identifier.urihttps://hdl.handle.net/20.500.12511/1975
dc.identifier.volume36
dc.identifier.wosqualityQ2
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoen
dc.publisherW B Saunders Co-Elsevıer Inc
dc.relation.ispartofAmerican Journal of Emergency Medicineen_US
dc.relation.publicationcategoryDiğer
dc.rightsinfo:eu-repo/semantics/embargoedAccess
dc.subjectAsprosin
dc.subjectBiochemical Marker
dc.subjectAngina Pectoris
dc.titleA novel biochemical marker for predicting the severity of ACS with unstable angina pectoris: Asprosin
dc.typeLetter

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