18F-FDG-PET correlates of ımpulse control disorder in a diabetic patient

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dc.authorid0000-0002-9704-6173
dc.authorid0000-0003-4292-5717
dc.authorid0000-0002-7685-2766
dc.authorid0000-0002-9815-1848
dc.contributor.authorYuluğ, Burak
dc.contributor.authorHanoğlu, Lütfü
dc.contributor.authorTavlı, Ahmet Mithat
dc.contributor.authorÇakır, Tansel
dc.contributor.authorKhanmammadov, Elmir
dc.contributor.authorOlmusçelik, Oktay
dc.date.accessioned10.07.201910:49:13
dc.date.accessioned2019-07-10T19:52:03Z
dc.date.available10.07.201910:49:13
dc.date.available2019-07-10T19:52:03Z
dc.date.issued2016
dc.departmentİstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, Nöroloji Ana Bilim Dalı
dc.departmentİstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, Nükleer Tıp Ana Bilim Dalı
dc.departmentİstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, İç Hastalıkları Ana Bilim Dalı
dc.descriptionWOS: 000397116200009
dc.descriptionPubMed ID: 27697074
dc.description.abstractBackground: Studies have already shown that hyperglycemia and insulin resistance are significantly associated with the impairment of cerebral glucose metabolism that may secondary lead to cognitive disturbances. In this study, we aimed to evaluate the neurometabolic correlates of diabetes in a patient with Intermittent explosive disorder (IED). Methods: We have investigated the cerebral glucose metabolism via 2-[18F]-fluoro-2-deoxy-D-glucose positron emission tomography (FDG-PET) in a diabetic patient with aggressive outbursts. Results: We have found significantly reduced glucose uptake in left temporoparietal region, pontin area, and left nucleus lentiformis. Discussion: Our present results indicate decreased cerebral glucose metabolism in specific cerebral cortical and subcortical areas. The main limitation of this report is that, this is a single case study and that these findings need to be replicated in well-conducted randomized controlled studies by using additional neuroquantitative methods.
dc.identifier.citationYuluğ, B., Hanoğlu, L., Tavlı, A. M., Çakır, T., Khanmammadov, E. ve Olmusçelik, O. (2016). 18F-FDG-PET correlates of ımpulse control disorder in a diabetic patient. Endocrine Metabolic & Immune Disorders-Drug Targets, 16(3), 224-227. https://dx.doi.org/10.2174/1872214810666160930124951
dc.identifier.doi10.2174/1872214810666160930124951
dc.identifier.endpage227
dc.identifier.issn1871-5303
dc.identifier.issn2212-3873
dc.identifier.issue3
dc.identifier.scopusqualityQ3
dc.identifier.startpage224
dc.identifier.urihttps://dx.doi.org/10.2174/1872214810666160930124951
dc.identifier.urihttps://hdl.handle.net/20.500.12511/2331
dc.identifier.volume16
dc.identifier.wosqualityQ3
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoen
dc.publisherBentham Scıence Publ Ltd
dc.relation.ispartofEndocrine Metabolic & Immune Disorders-Drug Targetsen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanı
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.subjectDiabetes Mellitus
dc.subjectFDG-PET
dc.subjectImpulse Control Disorder
dc.title18F-FDG-PET correlates of ımpulse control disorder in a diabetic patient
dc.typeArticle

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