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dc.contributor.authorMahroum, Naim
dc.contributor.authorShoenfeld, Yehuda
dc.date.accessioned2023-08-11T07:17:23Z
dc.date.available2023-08-11T07:17:23Z
dc.date.issued2023en_US
dc.identifier.citationMahroum, N. ve Shoenfeld, Y. (2023). SARS-CoV-2 infection provoking autoimmunity. Nephrology, Dialysis, Transplantation, 38(8), 1782-1784. https://dx.doi.org/10.1093/ndt/gfad077en_US
dc.identifier.issn0931-0509
dc.identifier.issn1460-2385
dc.identifier.urihttps://dx.doi.org/10.1093/ndt/gfad077
dc.identifier.urihttps://hdl.handle.net/20.500.12511/11304
dc.description.abstractFor decades, infections have been recognized as strong stimulators of the immune system and subsequently serve as a trigger for autoimmunity and autoimmune diseases. While infection is a very broad term, infectious agents such as bacteria, viruses and parasites, but particularly viruses present a key player in this regard and have been considered a classical example for this correlation.en_US
dc.language.isoengen_US
dc.publisherOxford University Pressen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectAutoimmunityen_US
dc.subjectInfectionen_US
dc.subjectSARS-CoV-2en_US
dc.titleSARS-CoV-2 infection provoking autoimmunityen_US
dc.typeeditorialen_US
dc.relation.ispartofNephrology, Dialysis, Transplantationen_US
dc.departmentİstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, İç Hastalıkları Ana Bilim Dalıen_US
dc.authorid0000-0002-7919-1326en_US
dc.identifier.volume38en_US
dc.identifier.issue8en_US
dc.identifier.startpage1782en_US
dc.identifier.endpage1784en_US
dc.relation.publicationcategoryDiğeren_US
dc.identifier.doi10.1093/ndt/gfad077en_US
dc.institutionauthorMahroum, Naim
dc.identifier.wosqualityQ1en_US
dc.identifier.wos001002567300001en_US
dc.identifier.scopus2-s2.0-85166383689en_US
dc.identifier.pmid37186258en_US
dc.identifier.scopusqualityQ1en_US


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