dc.contributor.author | Mahroum, Naim | |
dc.contributor.author | Shoenfeld, Yehuda | |
dc.date.accessioned | 2023-08-11T07:17:23Z | |
dc.date.available | 2023-08-11T07:17:23Z | |
dc.date.issued | 2023 | en_US |
dc.identifier.citation | Mahroum, N. ve Shoenfeld, Y. (2023). SARS-CoV-2 infection provoking autoimmunity. Nephrology, Dialysis, Transplantation, 38(8), 1782-1784. https://dx.doi.org/10.1093/ndt/gfad077 | en_US |
dc.identifier.issn | 0931-0509 | |
dc.identifier.issn | 1460-2385 | |
dc.identifier.uri | https://dx.doi.org/10.1093/ndt/gfad077 | |
dc.identifier.uri | https://hdl.handle.net/20.500.12511/11304 | |
dc.description.abstract | For decades, infections have been recognized as strong stimulators of the immune system and subsequently serve as a trigger for autoimmunity and autoimmune diseases. While infection is a very broad term, infectious agents such as bacteria, viruses and parasites, but particularly viruses present a key player in this regard and have been considered a classical example for this correlation. | en_US |
dc.language.iso | eng | en_US |
dc.publisher | Oxford University Press | en_US |
dc.rights | info:eu-repo/semantics/openAccess | en_US |
dc.subject | Autoimmunity | en_US |
dc.subject | Infection | en_US |
dc.subject | SARS-CoV-2 | en_US |
dc.title | SARS-CoV-2 infection provoking autoimmunity | en_US |
dc.type | editorial | en_US |
dc.relation.ispartof | Nephrology, Dialysis, Transplantation | en_US |
dc.department | İstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, İç Hastalıkları Ana Bilim Dalı | en_US |
dc.authorid | 0000-0002-7919-1326 | en_US |
dc.identifier.volume | 38 | en_US |
dc.identifier.issue | 8 | en_US |
dc.identifier.startpage | 1782 | en_US |
dc.identifier.endpage | 1784 | en_US |
dc.relation.publicationcategory | Diğer | en_US |
dc.identifier.doi | 10.1093/ndt/gfad077 | en_US |
dc.institutionauthor | Mahroum, Naim | |
dc.identifier.wosquality | Q1 | en_US |
dc.identifier.wos | 001002567300001 | en_US |
dc.identifier.scopus | 2-s2.0-85166383689 | en_US |
dc.identifier.pmid | 37186258 | en_US |
dc.identifier.scopusquality | Q1 | en_US |