Basit öğe kaydını göster

dc.contributor.authorÖzcan Çetin, Elif Hande
dc.contributor.authorKorkmaz, Ahmet
dc.contributor.authorKara, Meryem
dc.contributor.authorMerovci, Idriz
dc.contributor.authorGöçer, Kemal
dc.contributor.authorAksu, Ekrem
dc.contributor.authorAbusaif, Suhaib
dc.contributor.authorOğuz, Ozan
dc.contributor.authorÖzeke, Özcan
dc.contributor.authorÇay, Serkan
dc.contributor.authorÖzcan, Fırat
dc.contributor.authorAras, Dursun
dc.contributor.authorTopaloğlu, Serkan
dc.date.accessioned2023-02-23T06:41:29Z
dc.date.available2023-02-23T06:41:29Z
dc.date.issued2023en_US
dc.identifier.citationÖzcan Çetin, E. H., Korkmaz, A., Kara, M., Merovci, I., Göçer, K., Aksu, E. ... Topaloğlu, S. (2023). Multiple wide QRS tachycardias in the same individual with ischemic cardiomyopathy. Journal of Cardiovascular Electrophysiology, 34(1), 241-245. https://doi.org/10.1111/jce.15776en_US
dc.identifier.issn1045-3873
dc.identifier.issn1540-8167
dc.identifier.urihttps://doi.org/10.1111/jce.15776
dc.identifier.urihttps://hdl.handle.net/20.500.12511/10511
dc.description.abstractA 67 ‐year‐old diabetic man with ischemic cardiomyopathy presented with a recurrent defibrillator shock. His electrocardiograms showed both narrow (NCT) Figure 1A) and wide complex tachycardias (WCT; Figure 1B). He experienced an anterior myocardial infarction (MI) 11 years earlier that caused the left ventricular ejection fraction to decrease to 25%, and subsequently, a coronary artery bypass surgery was performed and, then implantable cardioverter‐defibrillator was implanted for primary prophylaxis 2 years ago. The patient underwent to electrophysiological study for his WCT with a prediagnosis of incessant ventricular tachycardia (VT). In the first stage, the His signals were poor; however, we noticed that the premature ventricular complexes (PVC) reset the subsequent cycle lengths (Figure 2). Since the WCT was right bundle branch block (RBBB) morphology; we applied premature atrial complexes (PAC) with (Figure 3A) and without (Figure3B) septal refractory. Interestingly, we noticed the change in the QRS morphology (Figure 4) and ventriculoatrial (V‐A; Figure 5) during ongoing WCT. What are the mechanism of the different QRS morphology and ventriculoatrial (V‐A) responses of this tachycardia?en_US
dc.language.isoengen_US
dc.publisherWileyen_US
dc.rightsinfo:eu-repo/semantics/embargoedAccessen_US
dc.subjectIschemic Cardiomyopathyen_US
dc.subjectNodofascicularen_US
dc.subjectNodoventricularen_US
dc.subjectVentricular Tachycardiaen_US
dc.subjectWide Complex Tachycardiaen_US
dc.titleMultiple wide QRS tachycardias in the same individual with ischemic cardiomyopathyen_US
dc.typeeditorialen_US
dc.relation.ispartofJournal of Cardiovascular Electrophysiologyen_US
dc.departmentİstanbul Medipol Üniversitesi, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, Kardiyoloji Ana Bilim Dalıen_US
dc.identifier.volume34en_US
dc.identifier.issue1en_US
dc.identifier.startpage241en_US
dc.identifier.endpage245en_US
dc.relation.publicationcategoryDiğeren_US
dc.identifier.doi10.1111/jce.15776en_US
dc.institutionauthorAras, Dursun
dc.identifier.wosqualityQ3en_US
dc.identifier.wos000898083900001en_US
dc.identifier.scopus2-s2.0-85144216112en_US
dc.identifier.pmid36511469en_US
dc.identifier.scopusqualityQ1en_US


Bu öğenin dosyaları:

Thumbnail

Bu öğe aşağıdaki koleksiyon(lar)da görünmektedir.

Basit öğe kaydını göster