18F-FDG-PET correlates of ımpulse control disorder in a diabetic patient
Erişim
info:eu-repo/semantics/closedAccessTarih
2016Yazar
Yuluğ, BurakHanoğlu, Lütfü
Tavlı, Ahmet Mithat
Çakır, Tansel
Khanmammadov, Elmir
Olmusçelik, Oktay
Üst veri
Tüm öğe kaydını gösterKünye
Yuluğ, B., Hanoğlu, L., Tavlı, A. M., Çakır, T., Khanmammadov, E. ve Olmusçelik, O. (2016). 18F-FDG-PET correlates of ımpulse control disorder in a diabetic patient. Endocrine Metabolic & Immune Disorders-Drug Targets, 16(3), 224-227. https://dx.doi.org/10.2174/1872214810666160930124951Özet
Background: Studies have already shown that hyperglycemia and insulin resistance are significantly associated with the impairment of cerebral glucose metabolism that may secondary lead to cognitive disturbances. In this study, we aimed to evaluate the neurometabolic correlates of diabetes in a patient with Intermittent explosive disorder (IED). Methods: We have investigated the cerebral glucose metabolism via 2-[18F]-fluoro-2-deoxy-D-glucose positron emission tomography (FDG-PET) in a diabetic patient with aggressive outbursts. Results: We have found significantly reduced glucose uptake in left temporoparietal region, pontin area, and left nucleus lentiformis. Discussion: Our present results indicate decreased cerebral glucose metabolism in specific cerebral cortical and subcortical areas. The main limitation of this report is that, this is a single case study and that these findings need to be replicated in well-conducted randomized controlled studies by using additional neuroquantitative methods.
WoS Q Kategorisi
Q3Scopus Q Kategorisi
Q3Kaynak
Endocrine Metabolic & Immune Disorders-Drug TargetsCilt
16Sayı
3Bağlantı
https://dx.doi.org/10.2174/1872214810666160930124951https://hdl.handle.net/20.500.12511/2331
Koleksiyonlar
- Makale Koleksiyonu [3642]
- PubMed İndeksli Yayınlar Koleksiyonu [4039]
- Scopus İndeksli Yayınlar Koleksiyonu [6274]
- WoS İndeksli Yayınlar Koleksiyonu [6417]